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      Targeted muscle reinnervation in the initial management of traumatic upper extremity amputation injury.

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          Abstract

          Targeted muscle reinnervation (TMR) was initially designed to provide cortical control of upper limb prostheses through a series of novel nerve transfers. Early experience has suggested that TMR may also inhibit symptomatic neuroma formation. We present the first report of TMR performed at the time of a traumatic shoulder disarticulation. The procedure was done to prevent painful neuroma pain and allow for myoelecteric prosthetic use in the future. Eight months post-operatively, the patient demonstrates multiple successful nerve transfers and exhibits no evidence of neuroma pain on clinical exam. Using the Patient Reported Outcomes Measurement Information System (PROMIS), the patient demonstrates minimal pain interference or pain behavior. Targeted muscle reinnervation may be considered in the acute trauma setting to prevent neuroma pain and to prepare patients for myoelectric prostheses in the future.

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          Author and article information

          Journal
          Hand (N Y)
          Hand (New York, N.Y.)
          Springer Nature America, Inc
          1558-9447
          1558-9447
          Jun 2014
          : 9
          : 2
          Affiliations
          [1 ] Division of Plastic and Reconstructive Surgery, Feinberg School of Medicine, Northwestern University, Chicago, IL USA.
          [2 ] Division of Plastic and Reconstructive Surgery, Feinberg School of Medicine, Northwestern University, Chicago, IL USA ; Plastic and Reconstructive Surgery, Neurosurgery, and Orthopedics, Northwestern Feinberg School of Medicine and Neural Engineering Center for Artificial Limbs, Chicago, IL USA.
          [3 ] Division of Plastic Surgery, Southern Illinois University School of Medicine, P.O. Box 19653, Springfield, IL 62794-9653 USA.
          Article
          9602
          10.1007/s11552-014-9602-5
          4022965
          24839430
          ed1d70e1-48b4-48e4-8097-7907f7e8033c
          History

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