Downregulation of the miR-30 family microRNAs contributes to endoplasmic reticulum stress in cardiac muscle and vascular smooth muscle cells.

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Abstract

Endoplasmic reticulum (ER) stress is a common subcellular response to stresses and central to ER stress is increased expression of glucose-regulated protein 78 (GRP78). However, the mechanisms for GRP78 upregulation remained poorly understood. Our study goal was to shed light on this issue.

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Author and article information

Journal

Journal ID (iso-abbrev): Int J Cardiol

Title: International journal of cardiology

Publisher: Elsevier BV

ISSN (Electronic): 1874-1754

ISSN (Print): 0167-5273

Publication date (Electronic): Apr 15 2014

Volume: 173

Issue: 1

Affiliations

[1 ] Heart Center, Beijing Chaoyang Hospital, Capital Medical University, No. 8 Gong-Ti South Road, Beijing 100020, PR China.

[2 ] Heart Center, Beijing Chaoyang Hospital, Capital Medical University, No. 8 Gong-Ti South Road, Beijing 100020, PR China. Electronic address: gbmiao.cmu@live.cn.

[3 ] Heart Center, Beijing Chaoyang Hospital, Capital Medical University, No. 8 Gong-Ti South Road, Beijing 100020, PR China. Electronic address: zhangjj.cmu@126.com.

Article

Publisher Item ID: S0167-5273(14)00360-X

DOI: 10.1016/j.ijcard.2014.02.007

PubMed ID: 24612558

SO-VID: 7ea37cd9-72d9-4d64-89e6-3f55afdcb84b

History

Keywords: Cardiac cell,ER stress,GRP78,Oxidative stress,Vascular smooth muscle cells,miR-30a

Data availability:

Keywords: Cardiac cell, ER stress, GRP78, Oxidative stress, Vascular smooth muscle cells, miR-30a

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