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Use of induced pluripotent stem cells to recapitulate pulmonary alveolar proteinosis pathogenesis.
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Abstract
In patients with pulmonary alveolar proteinosis (PAP) syndrome, disruption of granulocyte/macrophage colony-stimulating factor (GM-CSF) signaling is associated with pathogenic surfactant accumulation from impaired clearance in alveolar macrophages.
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