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Post-traumatic inflammation following spinal cord injury.

Publication date: 2003-06-30

Keywords: Animals, Calcium, metabolism, Cell Death, Free Radicals, adverse effects, Glial Fibrillary Acidic Protein, Glutamic Acid, Humans, Inflammation, etiology, Mitochondria, Neuroglia, Neurons, Neurotoxins, Nitric Oxide, Spinal Cord Injuries, pathology, physiopathology, Wallerian Degeneration

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Abstract

Inflammatory reaction following a spinal cord injury (SCI) contributes substantially to secondary effects, with both beneficial and devastating effects. This review summarizes the current knowledge concerning the structural features (vascular, cellular, and biochemical events) of SCI and gives an overview of the regulation of post-traumatic inflammation.

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PubMed ID:: 12815368

DOI:: 10.1038/sj.sc.3101483

ScienceOpen disciplines: Chemistry

Keywords: Animals,Calcium,metabolism,Cell Death,Free Radicals,adverse effects,Glial Fibrillary Acidic Protein,Glutamic Acid,Humans,Inflammation,etiology,Mitochondria,Neuroglia,Neurons,Neurotoxins,Nitric Oxide,Spinal Cord Injuries,pathology,physiopathology,Wallerian Degeneration

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ScienceOpen disciplines: Chemistry

Post-traumatic inflammation following spinal cord injury.

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