Toll-interacting protein (Tollip) negatively regulates pressure overload-induced ventricular hypertrophy in mice.

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Abstract

Toll-interacting protein (Tollip) is a critical regulator of the Toll-like receptor-mediated signalling pathway. However, the role of Tollip in chronic pressure overload-induced cardiac hypertrophy remains unclear. This study aimed to determine the functional significance of Tollip in the regulation of aortic banding-induced cardiac remodelling and its underlying mechanisms.

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Author and article information

Journal

Journal ID (iso-abbrev): Cardiovasc. Res.

Title: Cardiovascular research

ISSN (Electronic): 1755-3245

ISSN (Print): 0008-6363

Publication date (Electronic): Jan 1 2014

Volume: 101

Issue: 1

Affiliations

[1 ] College of Life Sciences, Wuhan University, Wuhan 430072, PR China.

Article

Publisher Item ID: cvt232

DOI: 10.1093/cvr/cvt232

PMC ID: 3968303

PubMed ID: 24285748

SO-VID: ee1e1aea-ba56-4ace-9faa-8290eec54342

History

Keywords: AKT,Cardiac remodelling,Cardiomyocyte hypertrophy,Pressure overload,Tollip

Data availability:

Keywords: AKT, Cardiac remodelling, Cardiomyocyte hypertrophy, Pressure overload, Tollip

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