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Molecular interplay between cdk4 and p21 dictates G0/G1 cell cycle arrest in prostate cancer cells.
2013-09-01
Cell Line, Tumor, Cell Survival, Cyclin-Dependent Kinase 4, genetics, metabolism, Cyclin-Dependent Kinase Inhibitor p21, Cyclin-Dependent Kinase Inhibitor p27, Dose-Response Relationship, Drug, E2F1 Transcription Factor, Furocoumarins, pharmacology, G0 Phase, drug effects, G1 Phase Cell Cycle Checkpoints, Gene Expression Regulation, Enzymologic, Gene Expression Regulation, Neoplastic, Humans, Male, Phosphorylation, Prostatic Neoplasms, enzymology, pathology, RNA Interference, Retinoblastoma Protein, Signal Transduction, Time Factors, Transcription, Genetic, Transfection
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Cell Line, Tumor,Cell Survival,Cyclin-Dependent Kinase 4,genetics,metabolism,Cyclin-Dependent Kinase Inhibitor p21,Cyclin-Dependent Kinase Inhibitor p27,Dose-Response Relationship, Drug,E2F1 Transcription Factor,Furocoumarins,pharmacology,G0 Phase,drug effects,G1 Phase Cell Cycle Checkpoints,Gene Expression Regulation, Enzymologic,Gene Expression Regulation, Neoplastic,Humans,Male,Phosphorylation,Prostatic Neoplasms,enzymology,pathology,RNA Interference,Retinoblastoma Protein,Signal Transduction,Time Factors,Transcription, Genetic,Transfection
Cell Line, Tumor, Cell Survival, Cyclin-Dependent Kinase 4, genetics, metabolism, Cyclin-Dependent Kinase Inhibitor p21, Cyclin-Dependent Kinase Inhibitor p27, Dose-Response Relationship, Drug, E2F1 Transcription Factor, Furocoumarins, pharmacology, G0 Phase, drug effects, G1 Phase Cell Cycle Checkpoints, Gene Expression Regulation, Enzymologic, Gene Expression Regulation, Neoplastic, Humans, Male, Phosphorylation, Prostatic Neoplasms, enzymology, pathology, RNA Interference, Retinoblastoma Protein, Signal Transduction, Time Factors, Transcription, Genetic, Transfection