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Orchestrating the unfolded protein response in health and disease.
2002-10-31
Activating Transcription Factor 6, Apoptosis, DNA-Binding Proteins, metabolism, Diabetes Mellitus, genetics, Endoplasmic Reticulum, Endoribonucleases, Enzyme Activation, Genetic Diseases, Inborn, Glucose, Humans, Hyperhomocysteinemia, Ischemia, Membrane Proteins, Models, Biological, Neoplasms, Organelles, Protein Folding, Protein-Serine-Threonine Kinases, RNA Splicing, Signal Transduction, Stress, Physiological, Transcription Factors, Virus Diseases, etiology, eIF-2 Kinase
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Activating Transcription Factor 6,Apoptosis,DNA-Binding Proteins,metabolism,Diabetes Mellitus,genetics,Endoplasmic Reticulum,Endoribonucleases,Enzyme Activation,Genetic Diseases, Inborn,Glucose,Humans,Hyperhomocysteinemia,Ischemia,Membrane Proteins,Models, Biological,Neoplasms,Organelles,Protein Folding,Protein-Serine-Threonine Kinases,RNA Splicing,Signal Transduction,Stress, Physiological,Transcription Factors,Virus Diseases,etiology,eIF-2 Kinase
Activating Transcription Factor 6, Apoptosis, DNA-Binding Proteins, metabolism, Diabetes Mellitus, genetics, Endoplasmic Reticulum, Endoribonucleases, Enzyme Activation, Genetic Diseases, Inborn, Glucose, Humans, Hyperhomocysteinemia, Ischemia, Membrane Proteins, Models, Biological, Neoplasms, Organelles, Protein Folding, Protein-Serine-Threonine Kinases, RNA Splicing, Signal Transduction, Stress, Physiological, Transcription Factors, Virus Diseases, etiology, eIF-2 Kinase