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      Unweaving the mitotic spindle: A focus on Aurora kinase inhibitors in lung cancer

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          Abstract

          Lung cancer is one of the most aggressive malignancies, classified into two major histological subtypes: non-small cell lung cancer (NSCLC), that accounts for about 85% of new diagnosis, and small cell lung cancer (SCLC), the other 15%. In the case of NSCLC, comprehensive genome sequencing has allowed the identification of an increasing number of actionable targets, which have become the cornerstone of treatment in the advanced setting. On the other hand, the concept of oncogene-addiction is lacking in SCLC, and the only innovation of the last 30 years has been the introduction of immune checkpoint inhibitors in extensive stage disease. Dysregulation of cell cycle is a fundamental step in carcinogenesis, and Aurora kinases (AURKs) are a family of serine/threonine kinases that play a crucial role in the correct advance through the steps of the cycle. Hyperexpression of Aurora kinases is a common protumorigenic pathway in many cancer types, including NSCLC and SCLC; in addition, different mechanisms of resistance to anticancer drugs rely on AURK expression. Hence, small molecule inhibitors of AURKs have been developed in recent years and tested in several malignancies, with different results. The aim of this review is to analyze the current evidences of AURK inhibition in lung cancer, starting from preclinical rationale to finish with clinical trials available up to now.

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          First-Line Atezolizumab plus Chemotherapy in Extensive-Stage Small-Cell Lung Cancer

          Enhancing tumor-specific T-cell immunity by inhibiting programmed death ligand 1 (PD-L1)-programmed death 1 (PD-1) signaling has shown promise in the treatment of extensive-stage small-cell lung cancer. Combining checkpoint inhibition with cytotoxic chemotherapy may have a synergistic effect and improve efficacy.
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            Durvalumab plus platinum–etoposide versus platinum–etoposide in first-line treatment of extensive-stage small-cell lung cancer (CASPIAN): a randomised, controlled, open-label, phase 3 trial

            Most patients with small-cell lung cancer (SCLC) have extensive-stage disease at presentation, and prognosis remains poor. Recently, immunotherapy has demonstrated clinical activity in extensive-stage SCLC (ES-SCLC). The CASPIAN trial assessed durvalumab, with or without tremelimumab, in combination with etoposide plus either cisplatin or carboplatin (platinum-etoposide) in treatment-naive patients with ES-SCLC.
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              Comprehensive genomic profiles of small cell lung cancer.

              We have sequenced the genomes of 110 small cell lung cancers (SCLC), one of the deadliest human cancers. In nearly all the tumours analysed we found bi-allelic inactivation of TP53 and RB1, sometimes by complex genomic rearrangements. Two tumours with wild-type RB1 had evidence of chromothripsis leading to overexpression of cyclin D1 (encoded by the CCND1 gene), revealing an alternative mechanism of Rb1 deregulation. Thus, loss of the tumour suppressors TP53 and RB1 is obligatory in SCLC. We discovered somatic genomic rearrangements of TP73 that create an oncogenic version of this gene, TP73Δex2/3. In rare cases, SCLC tumours exhibited kinase gene mutations, providing a possible therapeutic opportunity for individual patients. Finally, we observed inactivating mutations in NOTCH family genes in 25% of human SCLC. Accordingly, activation of Notch signalling in a pre-clinical SCLC mouse model strikingly reduced the number of tumours and extended the survival of the mutant mice. Furthermore, neuroendocrine gene expression was abrogated by Notch activity in SCLC cells. This first comprehensive study of somatic genome alterations in SCLC uncovers several key biological processes and identifies candidate therapeutic targets in this highly lethal form of cancer.
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                Author and article information

                Contributors
                Journal
                Front Oncol
                Front Oncol
                Front. Oncol.
                Frontiers in Oncology
                Frontiers Media S.A.
                2234-943X
                27 October 2022
                2022
                : 12
                : 1026020
                Affiliations
                [1] 1 Comprehensive Cancer Center, Fondazione Policlinico Universitario Agostino Gemelli, IRCCS , Rome, Italy
                [2] 2 Section of Medical Oncology, Università Cattolica del Sacro Cuore , Rome, Italy
                Author notes

                Edited by: Alessandro Morabito, Division of Experimental Thoraco Pulmonary Oncology (IRCCS), Italy

                Reviewed by: Alessandra Bearz, Department of Medical Oncology (IRCCS), Italy; Umberto Malapelle, University of Naples Federico II, Italy

                *Correspondence: Emilio Bria, emilio.bria@ 123456unicatt.it

                †These authors share last authorship

                This article was submitted to Thoracic Oncology, a section of the journal Frontiers in Oncology

                Article
                10.3389/fonc.2022.1026020
                9647054
                36387232
                c59af1db-c77e-4d2a-9fe6-b57b3abb1927
                Copyright © 2022 Stefani, Piro, Schietroma, Strusi, Vita, Fiorani, Barone, Monaca, Sparagna, Valente, Ferrara, D’Argento, Di Salvatore, Carbone, Tortora and Bria

                This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

                History
                : 23 August 2022
                : 17 October 2022
                Page count
                Figures: 0, Tables: 3, Equations: 0, References: 93, Pages: 11, Words: 5865
                Funding
                Funded by: Associazione Italiana per la Ricerca sul Cancro , doi 10.13039/501100005010;
                Categories
                Oncology
                Review

                Oncology & Radiotherapy
                lung,cancer,oncology,aurora kinase (aurk),mitosis,aurk inhibitors
                Oncology & Radiotherapy
                lung, cancer, oncology, aurora kinase (aurk), mitosis, aurk inhibitors

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