Periconceptional alcohol consumption causes fetal growth restriction and increases glycogen accumulation in the late gestation rat placenta.

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Abstract

Alcohol consumption is a common social practice among women of childbearing age. With 50% of pregnancies being unplanned, many embryos are exposed to alcohol prior to pregnancy recognition and formation of the placenta. The effects of periconceptional (PC) alcohol exposure on the placenta are unknown.

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Author and article information

Journal

Journal ID (iso-abbrev): Placenta

Title: Placenta

Publisher: Elsevier BV

ISSN (Electronic): 1532-3102

ISSN (Print): 0143-4004

Publication date (Electronic): Jan 2014

Volume: 35

Issue: 1

Affiliations

[1 ] School of Biomedical Sciences, The University of Queensland, St Lucia, Queensland 4072, Australia.

[2 ] The Department of Physiology, The University of Melbourne, Parkville, Victoria 3010, Australia.

[3 ] School of Biomedical Sciences, The University of Queensland, St Lucia, Queensland 4072, Australia. Electronic address: k.moritz@uq.edu.au.

Article

Publisher Item ID: S0143-4004(13)00781-9

DOI: 10.1016/j.placenta.2013.10.008

PubMed ID: 24239160

SO-VID: bd6d6a39-e0cb-4f3b-825a-3cba26633fae

History

Keywords: Ethanol,Glycogen,Periconceptional,Placenta,Preimplantation

Data availability:

Keywords: Ethanol, Glycogen, Periconceptional, Placenta, Preimplantation

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