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Abstract
A novel strategy was utilised to assess the risk to health from exposure to polycyclic
aromatic hydrocarbons (PAHs). Ambient monitoring was carried out by personal sampling.
Urinary thioethers (UTh) and urinary 1-hydroxypyrene (1-HP) were utilised for biological
monitoring. Urinary d-glucaric acid (UDGA) and sister chromatid exchange (SCE) in
peripheral blood lymphocytes were used as biological effect markers. The population
was categorised into exposed and control groups according to the external dose of
PAHs. The excretion of 1-HP in the controls over the 3-day period showed a relatively
stable baseline, while the exposed showed a significant increase over the same period
of time. SCE frequency in the exposed population was significantly different from
controls.