Exosome miR-30a-5p Regulates Glomerular Endothelial Cells&apos; EndMT and Angiogenesis by Modulating Notch1/VEGF Signaling Pathway.

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Abstract

Diabetic nephropathy (DN) is one of the microvascular complications of diabetes. Endothelial-mesenchymal transition (EndMT) and endothelial damage lead to abnormal angiogenesis in DN.

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Author and article information

Journal

Journal ID (iso-abbrev): Curr Gene Ther

Title: Current gene therapy

Publisher: Bentham Science Publishers Ltd.

ISSN (Electronic): 1875-5631

ISSN (Print): 1566-5232

Publication date (Electronic): 2024

Volume: 24

Issue: 2

Affiliations

[1 ] Department of Nephrology, Second Hospital of Lanzhou University, Lanzhou 730030, Gansu, China.

Article

Publisher Item ID: CGT-EPUB-134660

DOI: 10.2174/0115665232258527230919071328

PubMed ID: 37767799

SO-VID: 4de0665e-cb09-4b41-8da7-b0deeba27406

History

Keywords: miR-30a-5p,Diabetic nephropathy,Notch1,angiogenesis,exosomes,glomerular endothelial cells.

Data availability:

Keywords: miR-30a-5p, Diabetic nephropathy, Notch1, angiogenesis, exosomes, glomerular endothelial cells.

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Exosome miR-30a-5p Regulates Glomerular Endothelial Cells' EndMT and Angiogenesis by Modulating Notch1/VEGF Signaling Pathway.

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