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MicroRNA-200c modulates epithelial-to-mesenchymal transition (EMT) in human colorectal cancer metastasis.
Keun Hur
1 ,
Yuji Toiyama ,
Masanobu Takahashi ,
Francesc Balaguer ,
Takeshi Nagasaka ,
Junichi Koike ,
Hiromichi Hemmi ,
Minoru Koi ,
C Richard Boland ,
Ajay Goel
Sep 2013
5-aminosalicylic acid (5-ASA),
Colorectal cancer,
DNA microsatellite instability,
EMT,
HNPCC syndrome,
abdominal surgery,
cancer,
cancer genetics,
cancer prevention,
cancer syndromes,
carcinogenesis,
cell biology,
chemotherapy,
colon carcinogenesis,
colorectal antral surgery,
familial adenomatous polyposis,
gastric cancer,
hepatic surgery,
juvenile polyposis,
metastasis,
methylation,
miR-200c,
molecular genetics,
non-steroidal
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Abstract
Distant metastasis is the major cause of cancer-related death in patients with colorectal cancer (CRC). Although the microRNA-200 (miR-200) family is a crucial inhibitor of epithelial-to-mesenchymal transition (EMT) in human cancer, the role of miR-200 members in the pathogenesis of metastatic CRC has not been investigated.
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Author and article information
5-aminosalicylic acid (5-ASA),Colorectal cancer,DNA microsatellite instability,EMT,HNPCC syndrome,abdominal surgery,cancer,cancer genetics,cancer prevention,cancer syndromes,carcinogenesis,cell biology,chemotherapy,colon carcinogenesis,colorectal antral surgery,familial adenomatous polyposis,gastric cancer,hepatic surgery,juvenile polyposis,metastasis,methylation,miR-200c,molecular genetics,non-steroidal
5-aminosalicylic acid (5-ASA), Colorectal cancer, DNA microsatellite instability, EMT, HNPCC syndrome, abdominal surgery, cancer, cancer genetics, cancer prevention, cancer syndromes, carcinogenesis, cell biology, chemotherapy, colon carcinogenesis, colorectal antral surgery, familial adenomatous polyposis, gastric cancer, hepatic surgery, juvenile polyposis, metastasis, methylation, miR-200c, molecular genetics, non-steroidal