STAT3 signaling pathway is involved in decitabine induced biological phenotype regulation of acute myeloid leukemia cells.

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Abstract

This study aimed to investigate the role of signal transduction and transcriptional activator STAT3 and relevant signaling pathway in the DAC regulated biological phenotype of AML cells.

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Journal

Journal ID (iso-abbrev): Am J Transl Res

Title: American journal of translational research

ISSN: 1943-8141

Publication date (Electronic): 2015

Volume: 7

Issue: 10

Affiliations

[1 ] Laboratory Center, Changzhou No. 2 People's Hospital, Affiliated Hospital of Nanjing Medical University No. 29 Xinglong Lane, Changzhou 213000, China.

[2 ] Department of Hematology, Changzhou No. 2 People's Hospital, Affiliated Hospital of Nanjing Medical University No. 188 Gehu Middle Road, Changzhou 213000, China.

[3 ] Department of Laboratory Medicine, Huizhou No. 3 People's Hospital, Affiliated Hospital of Guangzhou Medical University No. 1 Xuebei Street, Qiaodong Road, Huizhou 615000, China.

Article

PMC ID: 4656766

PubMed ID: 26692933

SO-VID: 11acef89-0b76-4b3f-89cd-3532e9ff92aa

History

Keywords: Decitabine,NKG2D ligand,SOCSs,STAT3,acute myeloid leukemia,natural killer cells

Data availability:

Keywords: Decitabine, NKG2D ligand, SOCSs, STAT3, acute myeloid leukemia, natural killer cells

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