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Bmi1 controls tumor development in an Ink4a/Arf-independent manner in a mouse model for glioma.
Olaf van Tellingen, Danielle Hulsman, Tessa Buckle, Sophia W M Bruggeman, Ellen Tanger, M Lohuizen, John Zevenhoven, Marleen Blom
2007-09-30
3T3 Cells, Animals, Astrocytes, metabolism, pathology, Brain Neoplasms, genetics, Cell Differentiation, Cell Proliferation, Cell Transformation, Neoplastic, Cells, Cultured, Cyclin-Dependent Kinase Inhibitor p16, deficiency, Gene Expression Regulation, Neoplastic, Glioblastoma, Mice, Mice, Inbred BALB C, Mice, Knockout, Mice, Nude, Mutation, Neoplasm Staging, Neoplasms, Experimental, Neurons, Nuclear Proteins, Phenotype, Polycomb Repressive Complex 1, Proto-Oncogene Proteins, Receptor, Epidermal Growth Factor, Repressor Proteins, Signal Transduction, Stem Cells, Time Factors, Transduction, Genetic
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3T3 Cells,Animals,Astrocytes,metabolism,pathology,Brain Neoplasms,genetics,Cell Differentiation,Cell Proliferation,Cell Transformation, Neoplastic,Cells, Cultured,Cyclin-Dependent Kinase Inhibitor p16,deficiency,Gene Expression Regulation, Neoplastic,Glioblastoma,Mice,Mice, Inbred BALB C,Mice, Knockout,Mice, Nude,Mutation,Neoplasm Staging,Neoplasms, Experimental,Neurons,Nuclear Proteins,Phenotype,Polycomb Repressive Complex 1,Proto-Oncogene Proteins,Receptor, Epidermal Growth Factor,Repressor Proteins,Signal Transduction,Stem Cells,Time Factors,Transduction, Genetic
3T3 Cells, Animals, Astrocytes, metabolism, pathology, Brain Neoplasms, genetics, Cell Differentiation, Cell Proliferation, Cell Transformation, Neoplastic, Cells, Cultured, Cyclin-Dependent Kinase Inhibitor p16, deficiency, Gene Expression Regulation, Neoplastic, Glioblastoma, Mice, Mice, Inbred BALB C, Mice, Knockout, Mice, Nude, Mutation, Neoplasm Staging, Neoplasms, Experimental, Neurons, Nuclear Proteins, Phenotype, Polycomb Repressive Complex 1, Proto-Oncogene Proteins, Receptor, Epidermal Growth Factor, Repressor Proteins, Signal Transduction, Stem Cells, Time Factors, Transduction, Genetic