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      Enhanced glycemic control, pancreas protective, antioxidant and hepatoprotective effects by umbelliferon-α-D-glucopyranosyl-(2(I) → 1(II))-α-D-glucopyranoside in streptozotocin induced diabetic rats.

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          Abstract

          The objective of the present study was to evaluate the effect of umbelliferon-α-D-glucopyranosyl-(2I → 1II)-α-D-glucopyranoside (UFD) from Aegle marmelos Corr. on serum glucose, lipid profile and free radical scavenging activity in normal and STZ (streptozotocin) induced diabetic rats.

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          Selenium: biochemical role as a component of glutathione peroxidase.

          J Rotruck, A. Pope, H E Ganther … (1973)
          When hemolyzates from erythrocytes of selenium-deficient rats were incubated in vitro in the presence of ascorbate or H(2)O(2), added glutathione failed to protect the hemoglobin from oxidative damage. This occurred because the erythrocytes were practically devoid of glutathione-peroxidase activity. Extensively purified preparations of glutathione peroxidase contained a large part of the (75)Se of erythrocytes labeled in vivo. Many of the nutritional effects of selenium can be explained by its role in glutathione peroxidase.
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            A simplified method of evaluating dose-effect experiments.

            J LITCHFIELD, F WILCOXON (1949)
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              Postprandial hyperglycemia and diabetes complications: is it time to treat?

              A Ceriello (2004)
              Increasing evidence suggests that the postprandial state is a contributing factor to the development of atherosclerosis. In diabetes, the postprandial phase is characterized by a rapid and large increase in blood glucose levels, and the possibility that the postprandial "hyperglycemic spikes" may be relevant to the onset of cardiovascular complications has recently received much attention. Epidemiological studies and preliminary intervention studies have shown that postprandial hyperglycemia is a direct and independent risk factor for cardiovascular disease (CVD). Most of the cardiovascular risk factors are modified in the postprandial phase in diabetic subjects and directly affected by an acute increase of glycemia. The mechanisms through which acute hyperglycemia exerts its effects may be identified in the production of free radicals. This alarmingly suggestive body of evidence for a harmful effect of postprandial hyperglycemia on diabetes complications has been sufficient to influence guidelines from key professional scientific societies. Correcting the postprandial hyperglycemia may form part of the strategy for the prevention and management of CVDs in diabetes.
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                Author and article information

                Journal
                Journal ID (iso-abbrev): Springerplus
                Title: SpringerPlus
                Publisher: Springer Science and Business Media LLC
                ISSN: 2193-1801
                ISSN (Print): 2193-1801
                Publication date (Electronic): 2013
                Volume: 2
                Affiliations
                [1 ] Department of Pharmaceutical Sciences, Faculty of Health Sciences, Sam Higginbottom Institute of Agriculture, Technology & Sciences, Allahabad, Uttar Pradesh 211007 India.
                [2 ] Sidharatha Institute of Pharmacy, Dehradun, Uttrakhand 248001 India.
                [3 ] Department of Phytochemisty & Pharmacognosy, Faculty of Pharmacy, Jamia Hamdard, New Delhi 110062 India.
                Article
                Publisher Item ID: 693
                DOI: 10.1186/2193-1801-2-639
                PMC ID: 3862866
                PubMed ID: 24349947
                SO-VID: 06aa50a5-3f73-488b-b51c-948176dec31c
                History

                Keywords: Antidiabetic,Antihyperlipidemic,Glibenclamide,Streptozotocin,Umbelliferon-α-D-glucopyranosyl-(2I → 1II)-α-D-glucopyranoside
                Data availability:
                Keywords: Antidiabetic, Antihyperlipidemic, Glibenclamide, Streptozotocin, Umbelliferon-α-D-glucopyranosyl-(2I → 1II)-α-D-glucopyranoside

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