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      Esophageal contractions, bolus transit and perception of transit after swallows of liquid and solid boluses in normal subjects.

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          Abstract

          Esophageal dysphagia is the sensation that the ingested material has a slow transit or blockage in its normal passage to the stomach. It is not always associated with motility or transit alterations.

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          Estimation in Generalized Linear Models with Random Effects

          Robert Schall (1991)
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            Dysphagia in patients with Chagas' disease.

            R O Dantas (1998)
            Some patients with Chagas' disease and apparent normal esophageal function complain of dysphagia. With the objective of investigating the esophageal motility of these patients we studied the esophageal contraction amplitude, duration, velocity, and lower esophageal sphincter (LES) pressure of 34 patients with a positive serologic test for Chagas' disease, normal radiologic esophageal examination, peristaltic contractions in the esophageal body, and complete LES relaxation. Fourteen patients complained of dysphagia and 20 had no symptoms. The results were compared with those of 22 healthy controls. We used the manometric method with continuous perfusion. In patients without dysphagia, the LES pressure (17.8 +/- 1.2 mmHg, mean +/- SEM) and distal esophageal amplitude (71.8 +/- 7.9 mmHg) were lower than those of control subjects (24.3 +/- 1.8 mmHg and 100. 4 +/- 10.6 mmHg, respectively). The velocity of peristaltic contractions was higher in patients than in controls, but there was no difference between patients with or without dysphagia. The duration of contraction in the distal esophagus was longer in patients with dysphagia (3.9 +/- 0.2 sec) than in patients without dysphagia (3.1 +/- 0.2 sec) and controls (3.2 +/- 0.2 sec). We conclude that dysphagia in patients with Chagas' disease and a nondilated esophagus with peristaltic contractions and complete LES relaxation is related to a longer duration of contractions in the middle and distal esophageal body.
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              A comparison of esophageal motility in response to bread swallows and water swallows.

              Jennifer J. P. Collins, Patrick Johnston, Emily Blackwell … (1993)
              Studies of esophageal manometry during eating have demonstrated abnormal motility in patients with dysphagia in whom standard water-swallow manometry was normal. However, there have been few concurrent motility studies making a direct comparison of food swallows with water swallows. This paper presents the results of such a study in 20 healthy volunteers. A comparison of bread swallows with water swallows revealed that both peristaltic amplitude in the proximal esophagus and peristaltic duration throughout the esophagus were significantly increased (p < 0.05). Peristaltic propagation velocity was significantly decreased in the proximal and mid-esophagus (p < 0.05). Percentages of nonconducted and nonperistaltic contractions were significantly increased (p < 0.05-0.001) during bread-swallow manometry. Therefore, the response of the normal esophagus to food has been shown to be different from its response to water swallows. In particular, the high percentage of nonpropagated swallows in normal subjects when eating indicates that the results of food manometry in patients with dysphagia must include wider limits of normality.
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                Author and article information

                Journal
                Journal ID (iso-abbrev): Arq Gastroenterol
                Title: Arquivos de gastroenterologia
                Publisher: FapUNIFESP (SciELO)
                ISSN (Electronic): 1678-4219
                ISSN (Print): 0004-2803
                Publication date (Electronic): Dec 2012
                Volume: 49
                Issue: 4
                Affiliations
                [1 ] Department of Medicine, Medical School of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, Brazil.
                Article
                Publisher Item ID: S0004-28032012000400004
                DOI: 10.1590/s0004-28032012000400004
                PubMed ID: 23329218
                SO-VID: c554ad5d-fe8e-41ec-8bc8-e459669628da
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