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      Mass function and dynamical study of the open clusters Berkeley 24 and Czernik 27

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          Abstract

          We present a \(UBVI\) photometric study of the open clusters Berkeley 24 (Be 24) and Czernik 27 (Cz 27). The radii of the clusters are determined as 2\farcm7 and 2\farcm3 for Be 24 and Cz 27, respectively. We use the Gaia Data Release 2 (GDR2) catalogue to estimate the mean proper motions for the clusters. We found the mean proper motion of Be 24 as \(0.35\pm0.06\) mas yr\(^{-1}\) and \(1.20\pm0.08\) mas yr\(^{-1}\) in right ascension and declination for Be 24 and \(-0.52\pm0.05\) mas yr\(^{-1}\) and \(-1.30\pm0.05\) mas yr\(^{-1}\) for Cz 27. We used probable cluster members selected from proper motion data for the estimation of fundamental parameters. We infer reddenings \(E(B-V)\) = \(0.45\pm0.05\) mag and \(0.15\pm0.05\) mag for the two clusters. Analysis of extinction curves towards the two clusters show that both have normal interstellar extinction laws in the optical as well as in the near-IR band. From the ultraviolet excess measurement, we derive metallicities of [Fe/H]= \(-0.025\pm0.01\) dex and \(-0.042\pm0.01\) dex for the clusters Be 24 and Cz 27, respectively. The distances, as determined from main sequence fitting, are \(4.4\pm0.5\) kpc and \(5.6\pm0.2\) kpc. The comparison of observed CMDs with \(Z=0.01\) isochrones, leads to an age of \(2.0\pm0.2\) Gyr and \(0.6\pm0.1\) Gyr for Be 24 and Cz 27, respectively. In addition to this, we have also studied the mass function and dynamical state of these two clusters for the first time using probable cluster members. The mass function is derived after including the corrections for data incompleteness and field star contamination. Our analysis shows that both clusters are now dynamically relaxed

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          A neuronal cell line that does not express either prion or doppel proteins.

          Prions have been extensively studied since they represent a new class of infectious agents, the pathogenic prion protein (PrPSc). However, a central question on the physiological function of the normal prion protein (PrPC) remains unresolved. A cell model which was previously established from Rikn mice (PrP-/-) remains problematic because of its ectopic expression of the doppel (Dpl) which may have a neurotoxic effect. Here we established neuronal cell lines from Zürich I (PrP-/-) which do not express Dpl protein and ICR mice (PrP+/+) by transfecting with plasmid encoding for the large T antigen of SV40. The transformed cells have shown neuronal characteristics and, thus, these cell lines may provide a useful model to explore the function of neuronal PrPC.
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            Author and article information

            Journal
            12 October 2018
            Article
            1810.05380
            f5dcc295-09b8-416b-8b08-b5445dd9374b

            http://arxiv.org/licenses/nonexclusive-distrib/1.0/

            History
            Custom metadata
            16 pages including 8 tables. 22 figures. Accepted by MNRAS
            astro-ph.SR astro-ph.GA

            Galaxy astrophysics,Solar & Stellar astrophysics
            Galaxy astrophysics, Solar & Stellar astrophysics

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