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Helicobacter pylori
Helicobacter pylori eradication for dyspepsia is clinically useful
other
Author(s):
D. Mcnamara
,
C. O’Morain
Publication date
(Print):
2000
Publisher:
Springer Netherlands
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When did Helicobacter first colonise humans?
Most cited references
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U. S. Householder survey of functional gastrointestinal disorders
Douglas Drossman
,
Zhiming Li
,
Eileen Andruzzi
…
(1993)
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Regression of primary low-grade B-cell gastric lymphoma of mucosa-associated lymphoid tissue type after eradication of Helicobacter pylori
A.C. Wotherspoon
,
T.C. Diss
,
Alexander Pan
…
(1993)
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An international association between Helicobacter pylori infection and gastric cancer
The EUROGAST Study Group
(1993)
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Book Chapter
Publication date (Print):
2000
Pages
: 427-433
DOI:
10.1007/978-94-011-3927-4_45
SO-VID:
c6450a5d-3ffa-439a-9c0a-f2c9d93679ca
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Book chapters
pp. 3
What are the biochemical and physiological implications of the new genetic information?
pp. 15
The urease system of Helicobacter pylori
pp. 25
The amphibiotic relationship of Helicobacter pylori and humans
pp. 31
Helicobacter pylori is pathogenic flora
pp. 37
Disease-specific Helicobacter pylori virulence factors: the role of cagA, vacA, iceA, babA2 alone or in combination
pp. 45
Factors associated with disappearance of Helicobacter pylori in the West
pp. 53
Factors associated with disappearance of Helicobacter pylori in the Far East
pp. 57
Differences in prevalence of Helicobacter pylori and disease outcomes according to race/environmental factors in Southeast Asia
pp. 73
Infection with Helicobacter heilmannii (formerly Gastrospirillum horminis): characterization, epidemiology and therapy
pp. 83
Hepatobiliary Helicobacters: recognized animal pathogens with suspected pathogenic potential in humans
pp. 105
Novel Helicobacter species in the intestine
pp. 115
Diagnosis of Helicobacter pylori infection: faecal antigen determination
pp. 123
Pitfalls in Helicobacter pylori diagnosis
pp. 141
Overview of immune and inflammatory changes due to Helicobacter infection
pp. 151
Interaction of Helicobacter pylori with gastric epithelium
pp. 155
Helicobacter pylori and the epithelial barrier: role of oxidative injury
pp. 169
Immuno-inflammatory response to Helicobacter pylori in children
pp. 175
Severity and reversibility of mucosal inflammation in children and adolescents infected with Helicobacter pylori
pp. 187
Elimination of Helicobacter pylori is dependent on a Th2 response
pp. 193
Elimination of Helicobacter pylori is not dependent on a Th2 cytokine response
pp. 197
The inflamatory activity in Helicobacter pylori infection is predominantly organism related
pp. 203
The inflammatory activity in Helicobacter pylori infection is predominantly host-related
pp. 213
Helicobacter pylori gastritis — a global view
pp. 221
Unusual forms of gastric inflammation and their relationship to Helicobacter pylori infection
pp. 229
Can atrophic gastritis be diagnosed in the presence of Helicobacter pylori infection?
pp. 239
Mechanisms involved in gastric atrophy
pp. 249
Intestinal metaplasia: types, mechanisms of origin, and role in gastric cancer histogenesis
pp. 255
Long-term proton pump inhibitor therapy accelerates the onset of atrophic gastritis in Helicobacter pylori -positive patients
pp. 269
Proton pump inhibitors do not accelerate the development of gastric atrophy in Helicobacter pylori gastritis
pp. 281
Autoimmune gastritis via mimicking does occur
pp. 289
Autoimmune gastritis and antigenic mimicking
pp. 295
Carditis and intestinal metaplasia of the cardia is reflux related
pp. 299
Carditis and cardia intestinal metaplasia are Helicobacter pylori -related
pp. 307
Is gastric metaplasia in Helicobacter pylori really gastric?
pp. 315
Extragastric manifestations of Helicobacter pylori — are they relevant?
pp. 327
Peptic ulcer disease — the transitional zones are important
pp. 339
What causes Helicobacter pylori -negative non-NSAID-related ulcers?
pp. 347
From the pump to the helix
pp. 373
Mechanisms involved in the development of hypochlorhydria and pangastritis in Helicobacter pylori infection
pp. 385
Effect of Helicobacter pylori infection on gastric acid control using proton pump inhibitors
pp. 391
Rebound acid hypersecretion after acid-suppressive therapy
pp. 397
Gastric consequences of proton pump inhibitor therapy and Helicobacter pylori eradication
pp. 411
Current concepts of dyspepsia: the role of the nervous system
pp. 421
How to explain outcome differences in dyspepsia studies
pp. 427
Helicobacter pylori eradication for dyspepsia is clinically useful
pp. 435
Dyspepsia is no indication for Helicobacter pylori eradication
pp. 443
Role of Helicobacter pylori infection in NSAID-associated gastropathy
pp. 453
Role of Helicobacter pylori in NSAID gastrophy: can H. pylori infection be beneficial?
pp. 461
Helicobacter pylori and non-steroidal anti-inflammatory drugs
pp. 469
The impact of a test-and-treat strategy for Helicobacter pylori : the United States perspective
pp. 475
Test-and-treat strategy in dyspepsia — the European perspective
pp. 483
The impact of the ‘test-and-treat’ strategies for Helicobacter pylori infection — an Asian perspective?
pp. 489
Rodent models for Helicobacter-induced gastric cancer
pp. 507
Helicobacter pylori and gastric cancer: the risk is real
pp. 513
Helicobacter pylori in gastric malignancy: role of oxidants, antioxidants and other co-factors
pp. 525
Gastric markers of pre-malignancy are not reversible
pp. 535
The case for the reversibility of gastric dysplasia/neoplasia
pp. 541
Evaluation of the long-term outcome of Helicobacter pylori -related gastric mucosa-associated lymphoid tissue (MALT) lymphoma
pp. 551
Current state-of-the-art management for Helicobacter pylori infection: global perspective
pp. 559
Guidelines for therapy of Helicobacter pylori infection — a world perspective
pp. 567
Bismuth triple and quadruple studies for Helicobacter pylori eradication in Canada
pp. 575
Approach to Helicobacter pylori infection in children
pp. 587
What role for clarithromycin in the treatment of Helicobacter pylori infection?
pp. 593
What is the role of bismuth in Helicobacter pylori antimicrobial resistance?
pp. 601
Risk factors for failure of Helicobacter pylori eradication therapy
pp. 609
Strategies for therapy failures: Choice of ‘back-up’ regimen determined by primary treatment for Helicobacter pylori infection
pp. 623
Quadruple should be first-line therapy for Helicobacter pylori infection
pp. 631
Quadruple therapy should be second-line treatment for Helicobacter pylori infection
pp. 637
Helicobacter pylori infection: expectations for future therapy
pp. 643
A Helicobacter pylori vaccine is essential
pp. 655
The agenda for the microbiologist
pp. 663
The agenda for the immunologist
pp. 669
The agenda for the histopathologist
pp. 673
Helicobacter infections in the new millennium: the challenge for the clinician
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